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Here are some links about retinal fibrosis.
This is from the above link.
Fibrosis commonly refers to the response of a tissue to injury. The injury can occur as a result of a mechanical wound or various metabolic malfunctions, including responses to inflammation, ischemia, and degenerative disease. The local response to such injuries includes infiltration by inflammatory cells, neovascularization, altered vascular permeability, proliferation of fibroblasts and fibroblast-like cells, modification of the ECM, and, ultimately, some sort of resolution of the damaged tissue. The CNS is highly specialized in many ways, including the types of inflammatory and wound-healing cells present. Since the retina is part of the CNS, its response to injury utilizes mechanisms very similar to those observed in the rest of the brain; this is true not only for the wound-healing response but also for utilization of migratory cues functional during development of the neuronal and vascular components of this highly organized tissue (1, 2). As discussed below, the response of the anterior segment of the eye to wound healing more closely resembles the response of non-CNS tissues than do such events in the posterior segment or the eye. Therefore, I refer to such wound-healing events in the anterior segment as fibrosis, whereas comparable events in the retina are referred to as gliosis. Although such distinction is somewhat artificial, it does serve to differentiate between the fibroblasts and glial cells that effect the wound-healing and scar-formation events.
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